If parietal cells are lost, mature chief cells do not form.

This is the major stimulus for acid secretion by parietal cells.

Another major effect is the loss of the intrinsic-factor-secreting parietal cells in the stomach lining.

There was a difference in the distribution of parietal cells between the fetus and infant specimens.

A canaliculus is an adaptation found on gastric parietal cells.

It works in conjunction with gastric chief cells and parietal cells.

Achlorhydria is another autoimmune disease of the parietal cells.

The damaged parietal cells are unable to produce the required amount of gastric acid.

Upon stimulation, adenylate cyclase is activated within the parietal cells.

We have shown, using immunohistochemistry, that intrinsic factor is present in parietal cells from 13 weeks of gestation.

The presence of serum antibodies to parietal cells and to intrinsic factor are characteristic findings.

Gastrin stimulates the production of gastric acid by parietal cells.

Parietal cells secrete acid in response to three types of stimuli:

This autoimmune disorder is localised to the body of the stomach, where parietal cells are located.

In five infant stomachs studied, however, extension of parietal cells into the antrum was much more limited.

Interestingly, this is the period in which parietal cells disappear from the same anatomical site in most subjects.

Parietal cells produce hydrochloric acid, a strong acid that helps to break down food.

All three of these stimulate parietal cells to secrete hydrochloric acid and intrinsic factor.

This may also be due to the autoimmune attack on the parietal cells of the stomach in pernicious anemia.

Gastrin acts on parietal cells directly and indirectly too, by stimulating the release of histamine.

Parietal cells: Produce hydrochloric acid and intrinsic factor.

Once present, acetylcholine activates G cells and parietal cells.

Histamine also stimulates H+ secretion from parietal cells.

Parietal cells also produce intrinsic factor.

There was great diversity in the density of parietal cells between specimens of different maturities (Fig 1).

The mediator for the action of ethanol on the parietal cell is not known.

If parietal cells are lost, mature chief cells do not form.

This is the major stimulus for acid secretion by parietal cells.

Another major effect is the loss of the intrinsic-factor-secreting parietal cells in the stomach lining.

There was a difference in the distribution of parietal cells between the fetus and infant specimens.

A canaliculus is an adaptation found on gastric parietal cells.

Further studies are clearly needed to fully elucidate the effect of ethanol on the gastric parietal cell.

Damage to or inhibition of the gastric parietal cell.

It works in conjunction with gastric chief cells and parietal cells.

Achlorhydria is another autoimmune disease of the parietal cells.

The damaged parietal cells are unable to produce the required amount of gastric acid.

Upon stimulation, adenylate cyclase is activated within the parietal cells.

We have shown, using immunohistochemistry, that intrinsic factor is present in parietal cells from 13 weeks of gestation.

This leads to protein loss, parietal cell atrophy, and an increase in mucous cells.

The presence of serum antibodies to parietal cells and to intrinsic factor are characteristic findings.

Gastrin stimulates the production of gastric acid by parietal cells.

Parietal cells secrete acid in response to three types of stimuli:

This autoimmune disorder is localised to the body of the stomach, where parietal cells are located.

Ethanol has a systemic as well as topical action on the gastric parietal cell.

Parietal cell and intrinsic factor blocking antibodies were not detected in any subject with atrophy.

In five infant stomachs studied, however, extension of parietal cells into the antrum was much more limited.

Interestingly, this is the period in which parietal cells disappear from the same anatomical site in most subjects.

Parietal cells produce hydrochloric acid, a strong acid that helps to break down food.

Parietal cell vagotomy: Surgery to cut the nerve that causes stomach cells to make acid.

All three of these stimulate parietal cells to secrete hydrochloric acid and intrinsic factor.