volume contraction

Both of these reactions are exothermic and proceeds with volume contraction.

There are several possible explanations for the process of alkalosis observed after volume contraction.

The second term, adjusts for the additional urea that is cleared from the body through volume contraction.

Volume contraction is sometimes even used synonymously with hypovolemia.

ICF volume contraction may cause disturbances in various organs throughout the body.

Volume contraction is a decrease in body fluid volume, with or without a concomitant loss of osmolytes.

Zintl noted that there was an atomic volume contraction when these compounds were formed and realised this could indicate cation formation.

Contraction alkalosis refers to the increase in blood pH that occurs as a result of fluid losses (volume contraction).

Reabsorption is also increased by volume contraction, reduced renal plasma flow as in congestive heart failure, and decreased glomerular filtration.

Volume contraction of intravascular fluid (blood plasma) is termed hypovolemia, and its signs include, in order of severity:

Then a loop diuretic is used, but only after the volume replacement is complete, otherwise volume contraction would result, which would further exacerbate the hypercalcemia.

The drying out of the surface layers would lead to a volume contraction; the hydrated inner layers resist this contraction, leading to surface stress cracking.

Extracellular fluid (ECF) volume contraction is associated with decreased blood volume and decreased renal perfusion pressure.

Treatment consists of NaCl infusion to correct ECF volume contraction and administration of K to replace urinary losses.

Volume contraction is more or less a loss of extracellular fluid (ECF) and/or intracellular fluid (ICF).

The method uses purely additive volume contributions for single atoms and additional correction factors for components with special functional groups which cause a volume contraction and therefore a higher density.

Volume contraction of extracellular fluid is directly coupled to and almost proportional to volume contraction of blood plasma, which is termed hypovolemia.

Volume contraction of intracellular fluid may occur after substantial fluid loss, since it is much larger than ECF volume, or loss of potassium (K) see section below.

In addition to urinary losses from polyuria and volume contraction, there is also obligate loss of potassium from kidney tubules as a cationic partner to the negatively charged ketone, β-hydroxybutyrate.

Finally, it has been suggested that the term "contraction alkalosis" is actually a misnomer, and that the alkalosis observed during volume contraction is actually attributable entirely to chloride depletion, which leads to a failure of pendrin, a chloride/bicarbonate transporter in the collecting duct.

It is thus the intravascular component of volume contraction (or loss of blood volume due to things such as hemorrhaging or dehydration), but, as it also is the most essential one, hypovolemia and volume contraction are sometimes used synonymously.

ECF volume contraction or hypovolemia is usually the type of volume contraction of primary concern in emergency, since ECF is approximately half the volume of ICF and is the first to be affected in e.g. bleeding.