ventral anterior nucleus

The ventral anterior nucleus is a nucleus of the thalamus.

The information is relayed to the medial dorsal and ventral anterior nuclei of the thalamus.

Damage to the mammillothalamic tract, ventral anterior nucleus, and ventral lateral nucleus can result in memory and language impairment.

The ventral anterior nucleus receives neuronal inputs from the basal ganglia although its main afferent fibres are from the globus pallidus.

It includes the ventral anterior nucleus and the medial part of the ventral lateral nucleus which receive projections primarily from the substantia nigra.

They provide connectivity between the globus pallidus and the thalamus, primarily the ventral anterior nucleus and the ventral lateral nucleus.

The fibers of this fasciculus then travel to the thalamus and primarily terminate in the ventral anterior nucleus and ventral lateral nucleus.

The ventral lateral nucleus in the thalamus forms the motor functional division in the thalamic nuclei along with the ventral anterior nucleus.

It is an output nucleus whose fibres project to thalamic nuclei, such as the ventral anterior nucleus, the ventrolateral nucleus, and the medial dorsal nucleus.

This may occur due to damage in the mammillary bodies, ventral anterior nucleus, and ventral lateral nucleus which has resulted in disruption in language in previous research.

There are significant projections to the thalamus (ventral lateral and ventral anterior nuclei), superior colliculus, and other caudal nuclei from the pars reticulata (the nigrothalamic pathway).

It originates in the cerebellar nuclei, crosses completely in the decussation of the brachium conjunctivum, bypasses the red nucleus, and terminates in parts of the ventral anterior nucleus, ventral intermediate, ventral posterolateral nucleus, and central lateral nuclei of the thalamus.

In a resting individual, a specific region of the globus pallidus, known as the internus, and a portion of the substantia nigra, known as the pars reticulata, send spontaneous inhibitory signals to the ventral anterior nucleus of the thalamus, through the release of GABA, an inhibitory neurotransmitter.