venous hypertension

Article: An unusual cause of venous hypertension after dialysis access creation.

This may be a sign of pulmonary venous hypertension.

Pulmonary venous hypertension is most often caused by congestive heart failure.

Pleural effusion may also be noticed, which is due to pulmonary venous hypertension.

Telangiectasia in the legs is often related to the presence of venous hypertension within underlying varicose veins.

Systemic venous hypertension can also increase the risk for developing hepatosplenomegaly, which may be seen in those patients with right-sided heart failure.

Pulmonary venous hypertension is exceedingly common, since it occurs in most patients symptomatic with congestive heart failure.

For example patients that have spinal dural fistulas can experience venous hypertension caused by thrombosis of these veins.

This causes pulmonary venous hypertension.

Increased blood pressure in the veins (venous hypertension) can cause diffusion of substances, including fibrin, out of capillaries.

They are thought to be due to venous hypertension caused by improper function of valves that exist in the veins to prevent blood from flowing backward.

An elevated JVP is the classic sign of venous hypertension (e.g. right-sided heart failure).

In such patients, neurological and visual deficit can be correlated to venous hypertension, from incorrect occipital venous emptying.

Pathogenesis in pulmonary venous hypertension (WHO Group II) is completely different.

Pulmonary Venous Hypertension: Increased blood pressure in the pulmonary veins (carrying blood away from the lungs, to the heart).

The venous hypertension also contributes to the head swelling seen in photos of astronauts and the nasal and sinus congestion along with headache noted by many.

Venous hypertension induces a rupture of small superficial veins, subcutaneous hemorrhage and an increase of tissue permeability.

Venous disease, such as venous incompetence, venous hypertension, and body mass (obesity) may be relevant to the underlying pathogenesis.

Total triterpenic fraction of Centella asiatica in the treatment of venous hypertension: a clinical, prospective, randomized trial using a combined microcirculatory model.

A damaged mitral valve in the heart (mitral stenosis or mitral regurgitation) may contribute to pulmonary venous hypertension.

Furthermore, the magnitude of exercise-induced pulmonary arterial, capillary and venous hypertension is reportedly similar in horses either with or without EIPH.

Acroangiodermatitis is a rare skin condition characterised by hyperplasia of pre-existing vasculature due to venous hypertension from severe chronic venous stasis.

Treatment of edema and increased capillary filtration in venous hypertension with total triterpenic fraction of Centella asiatica: a clinical, prospective, placebo-controlled, randomized, dose-ranging trial.

This then causes a cascade of cranial venous hypertension, which decreases CSF resorption from the arachnoid granulations, leading to intracranial hypertension and papilledema.

Microcirculatory effects of total triterpenic fraction of Centella asiatica in chronic venous hypertension: measurement by laser Doppler, TcPO2-CO2, and leg volumetry.