vascularization
vascularisation

It is possible, though, that these changes simply reflect vascularization effects.

The level of vascularization in the pineal gland is high.

The vascularization pattern is similar to that of perionychium.

The large surface area and vascularization of the crest also suggests a thermoregulatory function.

This is because tumor growths require vascularization before they can develop more rapidly.

Vascularization is not seen in any other crocodilian, and may have been unique to Aegisuchus.

After implant vascularization an optional secondary procedure can be done to place a couple peg or post.

Pathological vascularization of the cornea cause it to turn pink or salmon colored.

The tumor has a scant stroma and central necrosis because of the poor vascularization.

Compromised vascularization and remodeling capacity of bone leads to risk for osteonecrosis.

Vascularization of the cornea is not present.

A subset of participants also experienced increases in cerebral vascularization similar to those witnessed upon increasing physical activity.

Furthermore, the amount of SOX9 is reduced and defects in vascularization are found.

In addition, the porous structure allows for soft tissue adherence and vascularization within the implant.

Moreover, defects in vascularization are found.

It is then repeated every 1-3 weeks until vascularization is complete (or until disease progression mandates treatment).

That procedure is usually delayed for at least 6 months after enucleation to allow time for implant vascularization.

In order to work properly, there is neither innervation nor vascularization within the central portion of the articular disc.

In a healthy breast, the pattern formed by a normal vascularization looks like a pointed image in the direction of the nipple.

So these two genes may contribute to both the lack of apoptosis and vascularization of gliomas.

It is a significant softening of the vaginal portion of the cervix from increased vascularization.

Developmentally it is involved in vascularization, longitudinal cell expansion and increased radial expansion.

The enzyme plays a role in endometrial menstrual breakdown, regulation of vascularization and the inflammatory response.

The absence or dysfunction of these cells is implicated in impaired vascularization in cardiac and diabetes patients.

This vascularization is a result of hypertrophy and engorgement of the vessels below the growing uterus.