myocyte

So in the standard myocyte model this phase will be an horizontal line.

The size of the osculum is regulated by the myocyte.

The cardiac myocyte is the major cell involved in remodeling.

As the heart fills with more blood than usual, there is an increase in the load experienced by each myocyte.

This means that the refractoriness of the ventricular myocyte increases at lower heart rates.

This in turn triggers contraction of a myocyte.

Calcium is an essential factor for the development of cardiac myocyte contraction and relaxation.

This family includes the Myf5 protein, which is responsible for directing cells to the skeletal myocyte lineage during development.

This provided a consistent histological section for estimates of myocyte vacuolization and toxicity.

The standard model used to understand the cardiac action potential is the action potential of the ventricular myocyte.

The diad is a structure in the cardiac myocyte located at the sarcomere Z-line.

The end-plate potential propagates throughout the myocyte's sarcolemma and into the T-tubule system.

Phase 1 of the myocyte action potential occurs with the inactivation of the fast Na channels.

Like skeletal myocytes (and axons for that matter), a given cardiac myocyte has a negative membrane potential when at rest.

The electrical depolarizations that trigger cardiac myocytes to contract arise spontaneously within the myocyte itself.

There is evidence of myocyte (myocardial cell) degeneration and death seen in 50% of cases of fatty infiltration.

The unique functional aspect of this type of myocyte is the absence of a stable resting potential during diastole.

The threshold potential is the potential an excitable cell membrane, such as a myocyte, must reach in order to induce an action potential.

Each myocyte contains myofibrils, which are very long chains of sarcomeres, the contractile units of the cell.

In ischemia, the major damage to the cardiac myocyte, due to hypoxia, is seen following the reperfusion of blood (Vie et al.,.

However, a direct cardiac myocyte toxicity from norepinephrine iswell recognized [ 6 7 ] .

A myocyte (also known as a muscle cell or muscle fiber) is the type of cell found in muscle tissue.

Preload is described as the stretching of a single cardiac myocyte immediately prior to contraction and is therefore related to the sarcomere length.

Inhibiting potassium channels, slowing repolarization, results in slowed atrial-ventricular myocyte repolarization.

Myocyte Enhancer Factor (2)