hyaloid

As the hyaloid vessels disappeared the number of macrophages declined.

The anterior hyaloid membrane separates the front of the vitreous from the lens.

The hyaloid fossa is a depression on the anterior surface of the vitreous body in which lies the lens.

The hyaloid artery provides nutrition to the lens during development in the fetus, and runs forward to the lens from the optic disc.

Regression of the hyaloid vasculature is mediated via macrophage-dependent cell death [ 35 ] .

In Bmp4 +/+mice the hyaloid vessels disappeared around P30, as reported elsewhere [ 33 34 ] .

The hyaloid artery, an artery running through the vitreous humour during the fetal stage of development, regresses in the third trimester of pregnancy.

The hyaloid artery is a branch of the ophthalmic artery, which is itself a branch of the internal carotid artery.

In wild type mice, the absolute number of hyaloid vessels actively decreases between P4 and P10 and all vessels disappear by P30.

Importantly, expression was not present in the developing trabecular meshwork and Schlemm's canal (ocular drainage structures) or in hyaloid vascular endothelial cells.

Occasionally, failure of the normal third trimester gestational atrophy of the hyaloid canal and the tunica vasculosa lentis is associated with other anomalies.

In the first ten days after birth, macrophages were abundant in the vitreous of Bmp4 +/+mice, usually localized close to the hyaloid vascular system (Figure 4F).

The vitreous membrane (or hyaloid membrane or vitreous cortex) is a layer of collagen separating the vitreous humour from the rest of the eye.

Regression of the hyaloid artery leaves a clear central zone through the vitreous humor, called the hyaloid canal or Cloquet's canal.

Syneresis: there is collapse of the vitreous due to collection of synchytic fluid between the posterior hyaloid membrane and the internal limiting membrane of the retina.

The posterior segment is the back two-thirds of the eye that includes the anterior hyaloid membrane and all structures behind it: the vitreous humor, retina, choroid, and optic nerve.

It refers to the separation of the posterior hyaloid membrane from the retina anywhere posterior to the vitreous base (a 3-4mm wide attachment to the ora serrata.)

Also sometimes called Abercrombie's disease, Abercrombie's syndrome, Virchow's syndrome, bacony disease, cellulose disease, hyaloid disease, lardaceous disease, and waxy disease.

In contrast, the hyaloid vessels in Bmp4 +/-heterozygotes persisted throughout the 17 month period studied and increased in number and size beyond that normally present at birth (Figure 4D,E,F,G).

Persistent hyperplastic primary vitreous (PHPV) is a rare congenital developmental anomaly of the eye that results following failure of the embryological, primary vitreous and hyaloid vasculature to regress.

Bmp4affects retinal vascular development and hyaloid vascular involution Bmp4 +/-heterozygotes have abnormally arranged retinal blood vessels and abnormal persistence of the hyaloid vasculature.

Intraocular hemorrhage (bleeding into the eyeball) may occur in response to the raised pressure: subhyaloid hemorrhage (bleeding under the hyaloid membrane, which envelops the vitreous body of the eye) and vitreous hemorrhage may be visible on fundoscopy.

The white fluffy material is seen in many tissues both ocular and extraocular: in the anterior chamber structures, trabecular meshwork, central disc, zonular fibres, anterior hyaloid membrane, pupillary and anterior iris, trabecula, and occasionally the cornea.

Hyaloid canal is a small transparent canal running through the vitreous body from the optical nerve disc to the lens; in the fetus it contains a prolongation of the central artery of the retina, the hyaloid artery.