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Influence of erythrocyte aggregation on in vivo blood flow is still a controversial issue.
It is aggravated by local platelet and erythrocyte aggregation.
Erythrocyte aggregation is the main determinant of blood viscosity at low shear rate.
Erythrocyte aggregation is determined by both suspending phase (blood plasma) and cellular properties.
Erythrocyte aggregation is a physiological phenomenon that takes places in normal blood under low-flow conditions or at stasis.
The presence or increased concentrations of acute phase proteins, particularly fibrinogen, results in enhanced erythrocyte aggregation.
Erythrocyte aggregation is the reversible clumping of these cells under low shear forces or at stasis.
Mechanism of erythrocyte aggregation and sedimentation.
Red blood cells have unique mechanical behavior, which can be discussed under the terms "erythrocyte deformability" and "erythrocyte aggregation".
Erythrocyte aggregation also affects hemodynamic mechanisms in microcirculation and vascular control mechanisms.
Effect of methoxylated flavones on erythrocyte aggregation and sedimentation in blood of normal subjects: Evidence of a dietary role for flavonoids.
The antithrombotic effect of dextran is mediated through its binding of erythrocytes, platelets, and vascular endothelium, increasing their electronegativity and thus reducing erythrocyte aggregation and platelet adhesiveness.
The finding that for small tubes with diameters below about 300 micrometres and for faster flow rates which do not allow appreciable erythrocyte aggregation, the effective viscosity of the blood depends on tube diameter is known as the Fåhraeus-Lindqvist Effect.
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