The composition of individual bile acids was similar between the two groups.

However, the regulatory potential of bile acids is only beginning to be realized.

The reaction known by his name for the detection of bile acids was published in 1844.

The mechanisms by which bile acids may produce these effects are uncertain.

Intragastric bile acids were measured in 55 of 88 patients.

High fat consumption increases the amount of bile acids in the colon.

Animal model studies support the concept that bile acids may play a role in the development of colon cancer.

Primary and secondary bile acids help the body digest fats.

Bile acids can damage cells and possibly cause cancer.

The lower detection limit for bile acids was 0.3 pmol.

In order to compensate for this, cholesterol from the liver may be used to make more bile acids.

It is most prominently involved in the biosynthesis of bile acids.

In comparison to the more common bile acids, they have additional hydroxyl groups.

Bile acids may be mutagenic, but some of the techniques used have been questioned.

Bile acids work slowly to dissolve gallstones made of cholesterol.

Bile acids can promote tumor growth, especially of the cells that line the colon.

That makes sense, Wong says, because the liver uses cholesterol to make bile acids.

There are additional secondary bile acids, such as ursodeoxycholic acid.

Once in the cell, the cholesterol can be broken down and excreted as bile acids.

Increased bacterial concentrations are also involved in the deconjugation of bile acids.

This protein functions as a cell surface receptor for bile acids.

Cholesterol is oxidized by the liver into a variety of bile acids.

The more fat that people eat, the more bile acids they secrete.

The finding that patients with almost normal fat excretion continue to lose large amounts of bile acids.

The composition of individual bile acids was similar between the two groups.

This medication works by removing bile acid from the body.

However, the regulatory potential of bile acids is only beginning to be realized.

The reaction known by his name for the detection of bile acids was published in 1844.

Bile acid is a natural substance the liver makes by using cholesterol.

The mechanisms by which bile acids may produce these effects are uncertain.

High fat consumption increases the amount of bile acids in the colon.

Primary and secondary bile acids help the body digest fats.

In order to compensate for this, cholesterol from the liver may be used to make more bile acids.

In comparison to the more common bile acids, they have additional hydroxyl groups.

Animal model studies support the concept that bile acids may play a role in the development of colon cancer.

Bile acids can promote tumor growth, especially of the cells that line the colon.

That makes sense, Wong says, because the liver uses cholesterol to make bile acids.

It is also likely to improve patient compliance in taking the daily quota of bile acid.

Once inside the cell, the cholesterol is broken down and excreted as bile acid.

Bile acids work slowly to dissolve gallstones made of cholesterol.

Bile acids can damage cells and possibly cause cancer.

This protein functions as a cell surface receptor for bile acids.

Cholesterol is oxidized by the liver into a variety of bile acids.

Emich started with work on natural products, for example bile acid.

The more fat that people eat, the more bile acids they secrete.

Intragastric bile acids were measured in 55 of 88 patients.

In the past, many patients were accepted for bile acid treatment without specific, gall stone related symptoms.

Both factors have been shown by several groups to be associated with changes in biliary bile acid composition.