Todd's paresis

The most significant issue regarding the Todd's paresis is its differentiation from a stroke.

Todd's paresis may also affect speech, eye position (gaze), or vision.

The cause of Todd's paresis is unknown, but there are two hypotheses to its cause.

An infant with Todd's paresis does not necessarily preclude the diagnosis of a febrile convulsion.

Todd's paresis, as defined as any motor deficit after seizure, occurs in 13% of all seizures.

Todd's paresis is more common after any clonic seizure activity, and particularly if generalized tonic-clonic seizures occur.

The classic presentation of Todd's paresis is a transient weakness of a hand, arm, or leg after partial seizure activity within that limb.

Todd's paresis (TP) is a temporary regional loss of function in whatever region just experienced the seizure, and its manifestation depends on where the seizure was located.

Todd's paresis or Todd's paralysis (or postictal paresis/paralysis, "after seizure") is focal weakness in a part of the body after a seizure.

They are thought to be caused by a different mechanism than Todd's paresis, and including paralysis of the contralateral limb, and rare genetic causes of hemiplegia and seizures.

This view is as a result of a recent study that showed the incidence of Todd's paresis to be in 0.4% of infants that have been diagnosed with a febrile convulsion.

A Todd's paresis in this context may overestimate the extent of neurological deficit due to the vascular process itself resulting in erroneous decisions with regards to acute stroke therapy such as thrombolysis.

Todd's paresis can last for 24 or 48 hours, and reversible neurological defects (typically short term memory) can last for months, suggesting that more permanent changes in neuron structure may take place following seizures.